Existing cancer drugs have potential to benefit thousands more patients, research shows
byInstitute of Cancer ResearchSF3B1 hotspot mutations confer sensitivity to PARP inhibition by eliciting a defective replication stress response. Credit:Nature Genetics(2023). DOI: 1
Updated on: January 16,2024
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Existing cancer drugs have potential to benefit thousands more patients, research shows
byInstitute of Cancer ResearchSF3B1 hotspot mutations confer sensitivity to PARP inhibition by eliciting a defective replication stress response. Credit:Nature Genetics(2023). DOI: 1
Updated on:January 16,2024
20
